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Pathology: T1:  isointense or hypointense T2:  heterogeneously hyperintense (Antoni type A: relatively low; Antoni type B: high) A-densely packed neural and fibrous tissue with little extra cellular fluid B-loosely arranged tissue with more extra cellular fluid LOCATION: intracranial schwannomas cranial nerves: although almost any cranial nerve may be involved, except olfactory nerves and optic nerves which lack sheaths composed of Schwann cells, by far the most commonly involved nerve is the  vestibulocochlear nerve (CN VIII)   non-cranial nerve or intracerebral (very rare)  intraorbital schwannoma: commonly arise from supraorbital and supratrochlear nerves in the upper anterior orbital cavity  10 spinal schwannoma arising from  spinal nerve roots trunk intercostal nerves : see  intercostal nerve schwannoma posterior mediastinum retroperitoneum gastrointestinal schwannoma limbs especially flexor surfaces (specifically ulnar and peroneal nerves)  29 year old female came for surgical ch

 H/o trauma at the age of 5yrs to left eye On usg- The globe is reduced in size (usually <20 mm) with a thickened/folded posterior sclera. Dystrophic calcification is common, and osseous  metaplasia  sometimes occurs, forming what is called "intraocular bone".  CT small and shrunken globe with foci of calcium deposits and ossification in the sclera, cornea, lens, retina, and optic nerve distortion of globe components with challenging to separate and identify structures fibrotic scarring with irregular globe contour and diffusely increased attenuation

This is an online E log book to discuss our patient's de-identified health data shared after taking his/her/guardian's signed informed consent.  Here we discuss our individual patient's problems through series of inputs from available global online community of experts with an aim to solve those patient's clinical problems with collective current best evidence based inputs.  This E log book also reflects my patient-centered online learning portfolio and your valuable inputs on the comment box is welcome.  Case presentation: A 28 yr old male pt was apparently asymptomatic 1month back.He observed that on long standing he developed swelling of both legs which relieved on rest. 12days back he had epigastric pain which was spasmodic type,no aggrevating and relieving factors,for which he had consulted a doctor who gave medications for his pain. 6days back- As the pain didn't get relieved with medications he consulted another doctor in bhuvangiri who had done ultrasound sc

This is an online E log book to discuss our patient's de-identified health data shared after taking his/her/guardian's signed informed consent.  Here we discuss our individual patient's problems through series of inputs from available global online community of experts with an aim to solve those patient's clinical problems with collective current best evidence based inputs.  This E log book also reflects my patient-centered online learning portfolio and your valuable inputs on the comment box is welcome.  Case presentation: 26yr old male k/c/o dm since 10yrs (diagnosed at the age of 15yrs with symptoms of polyuria, polydipsia), HTN since 6yrs  was apparently normal 1yr back  developed nausea and vomiting  5-6ep/day,non projectile,non bilious,watery for which he had consulted a doctor at nalgonda hospital,he had given him some medications and it was relieved. He had similar complaints later on which lasted for few days till 4months back ,came to our hospital with these c

1) diabetic kidney disease with anemia due to chronic kidney disease  2).reasons for   • azotemia-elevated glucose levels  increase the speed of blood flow into the kidney, lowering the filtration time .  • anemia- reduced erythropoietin levels  Low EPO levels cause  red blood cell count to drop and anemia to develop.  • hypoalbuminemia-changes in podocytes effacement cause albumin to filter leading to hypoalbuminemia In glomeruli, there is mesangial expansion, thickening of the basement membrane, and, characteristically, nodular glomerulosclerosis  • acidosis-Healthy kidneys remove acid from the body through urine and they keep the right amount of bicarbonate (base) in the blood. But in CKD, the kidneys can’t remove enough acid, which can lead to metabolic acidosis For people with CKD, metabolic acidosis is defined as persistently low bicarbonate levels of less than 22 mEq/L in the blood.                         3) Rationale : syp potchlor was given because of the hypokalemia.. Inj. N

This is an online E log book to discuss our patient's de-identified health data shared after taking his/her/guardian's signed informed consent.  Here we discuss our individual patient's problems through series of inputs from available global online community of experts with an aim to solve those patient's clinical problems with collective current best evidence based inputs.  This E log book also reflects my patient-centered online learning portfolio and your valuable inputs on the comment box is welcome.  Case presentation: 45 yr old lady who is a housewife and K/C/O DM 2 since 5yrs and HTN since 1yr,she  was apparently normal 6months back,used to develop pedal edema on and off  aggravates on walking and relieves on taking rest for which she was taken to nalgonda hospital and the doctor had advised her to decrease the fluid intake and gave some medications.It was associated with shortness of breath grade 3. 5days back she had developed pedal edema which got progressed a